Testosterone injection half life

My husband is now 50. His low-t set in about 3-3 1/2 years ago while he was deployed to Afghanistan. The doctors at the VA assumed it was just depression so they put him on an SSRI when he returned and also prescribed Viagra. They also checked his t-levels at that time and said they were “normal”. His libido tanked. Not good for me at all. I’m 9 years younger. When I found out that the SSRI could be to blame for his low libido he went back to the VA and switched meds. A year later it had not returned and he had also developed sleep apnea and was gaining weight. His mood was also very different and low. He was basically a completely different person. They checked his t-levels again, at my insistence, and again said they were “normal”. He retired in Jan 2014. By Jan 2015 the problem had not changed at all and he decided to see a GP. She had his numbers checked and said he was low, a 250. It frustrates me that the VA did not catch this. February 2015, he started using Androgel. At the end of June 2015 there was still no change and his numbers had actually dropped to a 235. He and the doctor decided to switch to injections. He gets a shot every 2 weeks. He had his third injection yesterday and still feels no different. My question… how long before he starts feeling different? Does the length of time we’ve been dealing with this matter? He is frustrated, wants to just give up on it. That breaks my heart because we aren’t as close as we were before.

Side effects of testosterone enanthate include symptoms of masculinization like acne , increased hair growth , voice changes , and increased sexual desire . [4] The drug is a synthetic androgen and anabolic steroid and hence is an agonist of the androgen receptor (AR), the biological target of androgens like testosterone and dihydrotestosterone (DHT). [7] [4] It has strong androgenic effects and moderate anabolic effects, which make it useful for producing masculinization and suitable for androgen replacement therapy . [4] Testosterone enanthate is a testosterone ester and a long-lasting prodrug of testosterone in the body. [6] [2] [3] Because of this, it is considered to be a natural and bioidentical form of testosterone. [8]

Treatment of hypogonadal men with Sustanon 250 results in a clinically significant rise of plasma concentrations of testosterone, dihydrotestosterone, estradiol and androstenedione, as well as decrease of SHBG (Sex hormone binding globulin). Luteinizing hormone (LH) and follicle-stimulating hormone (FSH) are restored to the normal range. In hypogonadal men, treatment with Sustanon 250 results in an improvement of testosterone deficiency symptoms. Moreover, treatment increases bone mineral density and lean body mass, and decreases body fat mass. Treatment also improves sexual function, including libido and erectile function. Treatment decreases serum LDL-C, HDL-C and triglycerides and increases haemoglobin and haematocrit, which may lead to polycythaemia. No clinically relevant changes in liver enzymes and PSA have been reported. Testosterone also produces systemic effects, such as increasing the retention of sodium, potassium and chloride leading to an increase in water retention. Treatment may result in an increase in prostate size, and worsening of lower urinary tract symptoms, but no adverse effects on prostate symptoms have been observed. In hypogonadal diabeteic patients, improvement of insulinsensitivity and/or reduction in blood glucose have been reported with the use of androgens. In boys with constitutional delay of growth and puberty, treatment with Sustanon 250 accelerates growth and induces development of secondary sex characteristics. In female-to-male transsexuals, treatment with Sustanon 250 induces masculinisation.

Testosterone is significantly correlated with aggression and competitive behaviour and is directly facilitated by the latter. There are two theories on the role of testosterone in aggression and competition. [78] The first one is the challenge hypothesis which states that testosterone would increase during puberty thus facilitating reproductive and competitive behaviour which would include aggression. [78] Thus it is the challenge of competition among males of the species that facilitates aggression and violence. [78] Studies conducted have found direct correlation between testosterone and dominance especially among the most violent criminals in prison who had the highest testosterone levels. [78] The same research also found fathers (those outside competitive environments) had the lowest testosterone levels compared to other males. [78]

Testosterone injection half life

testosterone injection half life

Testosterone is significantly correlated with aggression and competitive behaviour and is directly facilitated by the latter. There are two theories on the role of testosterone in aggression and competition. [78] The first one is the challenge hypothesis which states that testosterone would increase during puberty thus facilitating reproductive and competitive behaviour which would include aggression. [78] Thus it is the challenge of competition among males of the species that facilitates aggression and violence. [78] Studies conducted have found direct correlation between testosterone and dominance especially among the most violent criminals in prison who had the highest testosterone levels. [78] The same research also found fathers (those outside competitive environments) had the lowest testosterone levels compared to other males. [78]

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